ICD-10-CM code I21.A1 is reported for a current MI due to demand ischemia. Ischemia is due to oxygen starvation to some or all of the heart.
When you look up the code I24.A1 for a Type 2 MI, the inclusions under the main term include MI due to demand ischemia, and also MI secondary to ischemic imbalance. There’s also a “code first” note for the underlying cause if known, and a direction to read the Official Guidelines for Coding and Reporting, section I.C.9.e5.
For example, a physician recently documented that a patient had elevated troponin, likely a Type 2 MI/demand ischemia in the setting of a hypertensive emergency. In this case, demand ischemia would be a CC, and Type 2 MI would affect the DRG assignment, but it wouldn’t add a CC/MCC.
Type 2 NSTEMI is coded to I24.8 --- It is an Supply/demand (type 2) mismatch myocardial infarction not a True MI.
Supply/demand ischemia can be either “demand ischemia” (no infarction) or “Type 2 MI” (infarction due to supply/demand mismatch). Because the troponins were within the normal range, this would not be classified as a Type 2 myocardial infarction.
Demand ischemia, reported with ICD-10-CM code I24. 8 (other forms of acute ischemic heart disease), refers to the mismatch between myocardial oxygen supply and demand, which is evidenced by the release of cardiac troponin.
Type 2 MI is defined as "myocardial infarction secondary to ischaemia due to either increased oxygen demand or decreased supply, e.g. coronary artery spasm, coronary embolism, anaemia, arrhythmias, hypertension or hypotension."
A: ICD-10-CM code I24. 8 would be used for demand ischemia where the patient did not have a current myocardial infarction (MI). This code also covers other forms of ischemic heart diseases.
Type 2 NSTEMI is defined as myocardial ischemia resulting from mismatched myocardial oxygen supply and demand that is not related to unstable coronary artery disease (CAD).
Demand ischemia can occur without a myocardial infarction (MI), so if there is no mention of an MI, a query might be appropriate. The condition is frequently confused with type 2 MIs, and clarification may be needed to code the diagnosis reflective of the condition.
For example, a physician recently documented that a patient had elevated troponin, likely a Type 2 MI/demand ischemia in the setting of a hypertensive emergency. In this case, demand ischemia would be a CC, and Type 2 MI would affect the DRG assignment, but it wouldn't add a CC/MCC.
ACS is caused by a sudden onset of cardiac tissue ischemia secondary to impaired blood flow. The precipitating event is blockage in the coronary arteries or a mismatch between the demand and supply of blood to cardiac tissue.
Most NSTEMI (65%-90%) are type 1 MI. Patients with type 2 MI have multiple comorbidities and causes of in-hospital mortality among these patients are not always CV-related.
Myocardial ischemia can lead to serious complications, including: Heart attack. If a coronary artery becomes completely blocked, the lack of blood and oxygen can lead to a heart attack that destroys part of the heart muscle. The damage can be serious and sometimes fatal.
The term supply-demand mismatch usually refers to type 2 myocardial infarctions (T2MI) in which cell death results from an imbalance between myocardial oxygen supply and demand.
There is always an underlying condition or disease process that causes the Type 2 MI. Ischemia means insufficient blood perfusion, and prolonged ischemia leads to infarction, i.e., cell death. When cells die and break down, they release their contents, including troponin, a heart-muscle protein.
Type 1 MI is myocardial necrosis, or cell death, caused by an anatomic blockage of blood flow for a prolonged period of time. This is usually due to atherosclerotic plaque and rupture or thrombosis, causing mechanical coronary artery obstruction. Type 2 MI is also cell death, but in a non-anatomic distribution due to generalized hypoperfusion, ...
A second Type 1 MI can either be reinfarction in the same anatomic distribution, as an extension of the first MI, or a patient can have another Type 1 MI in a different vessel , with a different area of the heart being affected. Treatment of myocardial infarction has always been informed by the desire to prevent death, reinfarction, ...
There is always an underlying etiology. The implication of a Type 2 MI is that it portends a worse prognosis for the causative condition.
Type 5 MI, related to coronary artery bypass grafting. If the troponin is elevated but it does not constitute a Type 2 MI, there are numerous ways to refer to it, such as troponinemia, troponin leak, and non-zero troponin.
Such is the case with troponin. In October 2012, the Third Universal Definition of Myocardial Infarction (TUDMI) was published by the American Heart Association, redefining myocardial infarction (MI).
Some people who have myocardial ischemia don't have any signs or symptoms (silent ischemia).
It is very important to classify the etiology of a troponin elevation and treat accordingly.