Demand ischemia should be reserved for when there is evidence of supply-demand mismatch causing ischemia without an elevated troponin above the 99th percentile. If the troponin is > 99th percentile the diagnosis is a Type 2 MI.
Demand ischemia describes myocardial ischemia primarily due to cardiac supply/demand mismatch rather than CAD. In other words, the supply of blood flow to the heart is not limited but is inadequate to match the increased oxygen demands of an increased workload on the heart.
Demand ischemia, reported with ICD-10-CM code I24. 8 (other forms of acute ischemic heart disease), refers to the mismatch between myocardial oxygen supply and demand, which is evidenced by the release of cardiac troponin.
Q: How would you code elevated troponin due to demand ischemia? A: I would code I24. 8 (other forms of acute ischemic heart disease).
DEMAND-MI (NCT03338504) is the first prospective observational cohort study that aimed to determine the mechanism of myocardial injury and role of coronary disease in patients with type 2 myocardial infarction.
Demand ischemia is a specific type of ischemia where the oxygen requirements of the myocardium are not being met due to some increased need. In pure demand ischemia, there is no stenosis in the coronary arteries, yet the volume of oxygen-containing blood is insufficient to meet the needs of the heart muscle.
Type 2 MI is defined as "myocardial infarction secondary to ischaemia due to either increased oxygen demand or decreased supply, e.g. coronary artery spasm, coronary embolism, anaemia, arrhythmias, hypertension or hypotension."
A physician query is appropriate for the possibility of Type 2 MI when the diagnosis of demand ischemia is associated with elevated troponins above 99th percentile range. It may be necessary to verify what the 99th percentile reference range is for your hospital's lab.
Specifically, myocardial injury is defined by at least 1 cardiac troponin concentration above the 99th percentile upper reference limit. Myocardial infarction is a form of myocardial injury but requires clinical evidence of acute myocardial ischemia.
ICD-10 code I25. 2 for Old myocardial infarction is a medical classification as listed by WHO under the range - Diseases of the circulatory system .
Patients who present with abdominal discomfort and shortness of breath may have their cardiac troponin level measured, which, if combined with other features such as ischemic ECG changes and symptoms, may lead to a type 2 MI diagnosis.
The term supply-demand mismatch usually refers to type 2 myocardial infarctions (T2MI) in which cell death results from an imbalance between myocardial oxygen supply and demand.
An MI is coded as acute for a period of four weeks following onset; after that, it is assigned code I25.2 (old MI). Codes in category I22 are also provided for a subsequent type 1 MI (STEMI or NSTEMI), defined as another MI occurring within four weeks of a previous (initial) MI. In this situation, a code from I21 is also assigned for the initial MI.
The patient is admitted to the hospital on June 1 and is diagnosed with acute myocardial infarction, unspecified ( ICD-10 code I21.9). On July 7, the provider sees the patient for a follow-up visit and the patient receives care related to the myocardial infarction.
Type 1 is the classic spontaneous MI, primarily due to coronary artery disease (CAD) with atherosclerotic plaque rupture, ulceration, fissuring, erosion, or dissection causing intraluminal thrombosis. Occasionally type 1 occurs in the absence of CAD with spontaneous thrombosis of a coronary artery (particularly in women). Type 1 includes Q-wave infarction, ST-elevation MI, and non-ST elevation MI.
Myocardial Infarction has defined six types of MI. The two most commonly encountered are type 1 (primarily due to CAD) and type 2 (primarily due to myocardial supply/demand mismatch). For these two types, MI is defined as myocardial necrosis identified by a rise and/or fall of cardiac biomarkers to or from a level greater than the 99th percentile of the upper reference limit.
Type 1 MI is myocardial necrosis, or cell death, caused by an anatomic blockage of blood flow for a prolonged period of time. This is usually due to atherosclerotic plaque and rupture or thrombosis, causing mechanical coronary artery obstruction. Type 2 MI is also cell death, but in a non-anatomic distribution due to generalized hypoperfusion, ...
There is always an underlying condition or disease process that causes the Type 2 MI. Ischemia means insufficient blood perfusion, and prolonged ischemia leads to infarction, i.e., cell death. When cells die and break down, they release their contents, including troponin, a heart-muscle protein.
A second Type 1 MI can either be reinfarction in the same anatomic distribution, as an extension of the first MI, or a patient can have another Type 1 MI in a different vessel , with a different area of the heart being affected. Treatment of myocardial infarction has always been informed by the desire to prevent death, reinfarction, ...