Disseminated superficial actinic porokeratosis (DSAP) L56.5 is a billable/specific ICD-10-CM code that can be used to indicate a diagnosis for reimbursement purposes. The 2019 edition of ICD-10-CM L56.5 became effective on October 1, 2018. This is the American ICD-10-CM version of L56.5 - other international versions of ICD-10 L56.5 may differ.
What is disseminated superficial actinic porokeratosis? Disseminated superficial actinic porokeratosis, or DSAP, is an inherited keratinisation disorder that causes discrete dry patches on the arms and legs. DSAP is a special type of inherited 'sunspot".
The tendency to DSAP is inherited as an autosomal dominant characteristic, which means on average half of the children of an affected parent will also have the tendency. The causative genes in porokeratosis have included the mevalonate pathway genes MVD, MVK, FDPS and PMVK.
DSAP is sometimes confused with multiple actinic keratoses, but actinic keratoses are more likely to arise on the face and hands and have a central scale rather than a peripheral scale. What is the treatment for DSAP?
Disseminated superficial actinic porokeratosis (DSAP) is a skin condition that causes dry, scaly patches.[8488] Symptoms include a large number of small, brownish patches with a distinctive border, found most commonly on sun-exposed areas of the skin (particularly the lower arms and legs).[8488][8489] DSAP usually ...
Disseminated superficial actinic porokeratosisL56. 5 - Disseminated superficial actinic porokeratosis (DSAP) | ICD-10-CM.
Disseminated superficial actinic porokeratosis, or DSAP, is an inherited keratinisation disorder that causes discrete dry patches on the arms and legs. DSAP is a special type of inherited 'sunspot". The name porokeratosis means scaly pore and is a misnomer as porokeratosis is not related to pores.
DSAP is more common on your arms and legs but may also affect skin that is damaged from the sun. DSAP is caused by an ultra-sensitivity to sunlight. A majority of people with DSAP inherit it from family genes. However, it is possible to develop the condition if you have a weak immune system.
ICD-10-CM Code for Disseminated superficial actinic porokeratosis (DSAP) L56. 5.
It is sometimes confused with actinic keratosis which is also caused by sun exposure (See Patient Information Leaflet on Actinic Keratoses); however, actinic keratosis is more likely to arise on the face and hands. DSAP is twice as likely to develop in women compared with men and is more common in lighter skin type.
Treatment options include the following.Topical diclofenac. Diclofenac is an NSAID that inhibits COX-2. ... Ingenol mebutate. ... Topical vitamin D analog. ... 5-fluorouracil. ... Imiquimod. ... Photodynamic therapy. ... Retinoids. ... Cryotherapy and other.More items...
DSAPAcronymDefinitionDSAPDynamic Situation Assessment and PredictionDSAPDefense Security Assistance ProgramDSAPData Service Access PointDSAPDNA Sequence Analysis Program11 more rows
In terms of appearance, DSAP is more extensive than other subtypes and appears as reddish and brown spots. These tend to appear symmetrically across a person's back, arms, legs, and shoulders. A variety of factors may cause DSAP to develop. The main cause seems to be exposure to ultraviolet (UV) light.
The development of disseminated superficial porokeratosis is occasionally observed in association with renal transplant, autoimmune diseases and various hematological disorders, suggesting a certain immunosuppression may trigger a widespread abnormal keratinization.
DSAP is usually without symptoms. The affected areas often feel dry and rough. However, exposure to sun can cause them to itch or sting and grow in size (still remaining small) and number.
The causative genes in porokeratosis have included the mevalonate pathway genes MVD, MVK, FDPS and PMVK. These result in decreased cholesterol in the affected areas of the skin.
The smallest DSAP lesion is a 1–3 mm conical papule, skin coloured, brownish-red or brown in colour. It is based around a hair follicle containing a keratotic (scaly) plug. Larger plaques have a sharp, slightly raised, keratotic ring, a fraction of a millimetre thick, with a diameter of 10 mm or more. The skin within the ring is thinned and mildly reddened or slightly brown, and a pale ring may be seen just within the ridge. The ridge itself is often a darker brown than the rest of the lesion. The central area is most often pale and smooth, but it may be red, scaly, dry, or have scaly follicular plugs.
There are several kinds of porokeratosis, and these can occur in family members or in the patient that has DSAP. DSAP is sometimes confused with multiple actinic keratoses, but actinic keratoses are more likely to arise on the face and hands and have a central scale rather than a peripheral scale.
If the DSAP has been induced by drug-induced immune suppression, withdrawal of the drug has been reported to result in remission of DSAP.
The development of squamous cell carcinoma (SCC) within a DSAP lesion is the main concern. This is uncommon (< 10% of individuals with DSAP develop SCC). However, many patients with DSAP have had significant exposure to the sun and may also have actinic keratoses and other forms of skin cancer (particularly basal cell carcinoma ). SCC presents as a solitary tender enlarging scaly or ulcerated plaque or nodule.