ICD-10 Code range (D55-D59), Hemolytic anemias contains ICD-10 codes for Anemia due to enzyme disorders, Thalassemia, Sickle-cell disorders, Other hereditary hemolytic anemias, Acquired hemolytic anemia Subscribe to Codify and get the code details in a flash.
Glucose-6-phosphate dehydrogenase (G6PD) deficiency without anemia 1 D50-D89#N#2021 ICD-10-CM Range D50-D89#N#Diseases of the blood and blood-forming organs and certain disorders involving the... 2 D75#N#ICD-10-CM Diagnosis Code D75#N#Other and unspecified diseases of blood and blood-forming organs#N#2016 2017 2018 2019... More ...
D50-D89 Diseases of the blood and blood-forming organs and certain disorders involving the immune mechanism D55.8 is a billable/specific ICD-10-CM code that can be used to indicate a diagnosis for reimbursement purposes.
Hemolytic anemia is a form of anemia due to hemolysis, the abnormal breakdown of red blood cells (RBCs), either in the blood vessels (intravascular hemolysis) or elsewhere in the human body (extravascular, but usually in the spleen ). It has numerous possible consequences, ranging from relatively harmless to life-threatening.
Names and CodesCondition:1Glucose-6-phosphate dehydrogenase deficiencyICD-9-CM Code:5282.2—Anemias due to disorders of glutathione metabolismICD-10-CM Code:6D55.0—Anemia due to glucose-6-phosphate dehydrogenase [G6PD] deficiency3 more rows•Sep 14, 2009
This destruction of red blood cells is called hemolysis. The most common medical problem associated with glucose-6-phosphate dehydrogenase deficiency is hemolytic anemia, which occurs when red blood cells are destroyed faster than the body can replace them.
Glucose-6-phosphate dehydrogenase (G6PD) deficiency is a genetic metabolic abnormality caused by deficiency of the enzyme G6PD. This enzyme is critical for the proper function of red blood cells: when the level of this enzyme is too low, red blood cells can break down prematurely (hemolysis).
In people with G6PD deficiency, either the red blood cells do not make enough G6PD or what they do make doesn't work as it should. Without enough G6PD to protect them, the red blood cells break apart. This is called hemolysis (hih-MOL-ih-sis).
Your doctor can diagnose G6PD deficiency by performing a simple blood test to check G6PD enzyme levels. Other diagnostic tests that may be done include a complete blood count, serum hemoglobin test, and a reticulocyte count. All these tests give information about the red blood cells in the body.
G6PD deficiency should be considered in neonates who develop hyperbilirubinemia within the first 24 hours of life, a history of jaundice in a sibling, bilirubin levels greater than the 95th percentile, and in Asian males.
Most individuals with glucose-6-phosphate dehydrogenase (G6PD) deficiency do not need treatment. However, they should be taught to avoid drugs and chemicals that can cause oxidant stress. Patients should also avoid broad beans (ie, fava beans). Favism occurs primarily in the Mediterranean variety of G6PD deficiency.
It plays a critical role in red blood cells, which carry oxygen from the lungs to tissues throughout the body. This enzyme helps protect red blood cells from damage and premature destruction.
See Additional Information. Levels of G6PD are higher in the newborn than they are in the adult. When high levels are seen in older patients, it invariably reflects the presence of a young red blood cell population with reticulocytosis.
The hallmark of glucose-6-phosphate dehydrogenase (G6PD) deficiency is red blood cell (RBC) destruction in response to oxidative stress.
G6PD deficiency is a genetic disorder in which the body doesn't have enough of an enzyme (chemical) called glucose-6-phosphate dehydrogenase (G6PD). G6PD is important in protecting red blood cells.