Apr 03, 2017 · The majority of the NCDs included are a result of feedback received from previous ICD-10 NCD CR7818, CR8109, CR8197, CR8691, & CR9087. Some are the result of revisions required to other NCD-related CRs released separately that included ICD-10 coding. Implementation date: 01/04/2016 Effective date: 10/1/2015. (CR9252)
Hyperbaric Oxygen Therapy (HBOT) Payable Diagnosis Codes 1 ICD-10 PCS Codes Description 5A05121 Extracorporeal Hyperbaric Oxygenation, Intermittent 5A05221 Extracorporeal Hyperbaric Oxygenation, Continuous ICD-10 Codes Description A48.0 Gas gangrene H34.11 Central retinal artery occlusion, right eye
ICD-10-PCS diagnosis codes: Code Description 5A05121 Extracorporeal Hyperbaric Oxygenation, Intermittent 5A05221 Extracorporeal Hyperbaric Oxygenation, Continuous The following ICD Diagnosis Codes are considered medically necessary when submitted with the CPT, HCPCS and/or ICD Procedure codes above if medical necessity criteria are met: ICD-10-CM Diagnosis …
Article MM10220, Hyperbaric Oxygen (HBO) Therapy (Section C, Topical Application of Oxygen) Article MM11134, International Classification of Diseases, 10th Revision (ICD-10) And Other Coding Revisions to National Coverage Determinations (NCDs) …
Hyperbaric oxygen (HBO) therapy is a modality in which the entire body is exposed to oxygen under increased atmospheric pressure. HBO therapy is a medical treatment that involves breathing in pure oxygen while inside a sealed chamber whose air pressure is significantly higher than normal atmospheric pressure. This increased air pressure helps supply your lungs with a greater amount of oxygen and, in turn, deliver more oxygen to tissues throughout your body.
The Medicare Advantage Policy Guideline documents are generally used to support UnitedHealthcare Medicare Advantage claims processing activities and facilitate providers’ submission of accurate claims for the specified services. The document can be used as a guide to help determine applicable:
Cancer Care Ontario’s evidence-based care advice report on “Hyperbaric Oxygen Therapy for the Treatment and Prevention of Radionecrosis and Other Radiation-Induced Injuries in Cancer Patients” (2013) did not mention radiation induced pulmonary fibrosis/ injury as an indication of HBOT.
Hyperbaric oxygen therapy (HBOT) is defined as systemic treatment in which the entire patient is placed inside a pressurized chamber and breathes 100% oxygen under a pressure greater than 1 atmosphere (atm). It is used to treat certain diseases and conditions that may improve when an increased partial pressure of oxygen is present in perfused tissues.
Kraft and colleagues (2016) stated that central airway stenosis (CAS) after lung transplantation has been attributed in part to chronic airway ischemia; however, little is known about the time course or significance of large airway hypoxia early after transplantation. These researchers evaluated large airway oxygenation and hypoxic gene expression during the 1st month following lung transplantation and their relation to airway complications. Subjects who received lung transplantation underwent endobronchial tissue oximetry of native and donor bronchi at 0, 3, and 30 days after transplantation (n = 11) and/or endobronchial biopsies (n = 14) at 30 days for real-time polymerase chain reaction (PCR) of hypoxia-inducible genes. Patients were monitored for 6 months for the development of transplant-related complications. Compared with native endobronchial tissues, donor tissue oxygen saturations (Sto2) were reduced in the upper lobes (74.1 ± 1.8% versus 68.8 ± 1.7%; p < 0.05) and lower lobes (75.6 ± 1.6% versus 71.5 ± 1.8%; p = 0.065) at 30 days post-transplantation. Donor upper lobe and subcarina Sto2 levels were also lower than the main carina (difference of -3.9 ± 1.5 and -4.8 ± 2.1, respectively; p < 0.05) at 30 days. Up-regulation of hypoxia-inducible genes VEGFA, FLT1, VEGFC, HMOX1, and TIE2 was significant in donor airways relative to native airways (all p < 0.05); VEGFA, KDR, and HMOX1 were associated with prolonged respiratory failure, prolonged hospitalization, extensive airway necrosis, and CAS (p < 0.05). The authors concluded that these findings implicated donor bronchial hypoxia as a driving factor for post-transplantation airway complications; strategies to improve airway oxygenation, such as bronchial artery re-anastomosis and HBOT merit clinical investigation.
Davis et al (2011) stated that cyclophosphamide-induced hemorrhagic cystitis (CHC) is an uncommon, but well-recognized condition caused by a metabolite, acrolein, which is toxic to the urothelium. Based on similarities in the histopathology of radiation- and chemotherapy-induced HC, benefit from HBOT has been proposed. Hyperbaric oxygen therapy produces an increased oxygen partial pressure diffusion gradient between the circulation and surrounding tissues, which enhances neutrophil function and fibroblast and macrophage migration into damaged hypoxic soft tissue, promoting collagen formation, fibroblast growth, angiogenesis and white-cell bacterial killing. There are only isolated case reports of HBOT for CHC, in the literature, thus, these investigators reviewed the New Zealand experience with HBOT in CHC. The case records of all patients with CHC referred to the 3 hyperbaric medicine units in New Zealand between 2000 and 2007 were reviewed retrospectively. A total of 6 patients, with life-threatening hemorrhage at the time of referral for HBOT weeks or months after initial presentation with CHC, were identified. Cessation of bleeding occurred in all 6 patients after 14 to 40 HBOT, without complications. All patients remained clear of hematuria at 11 to 36 months follow-up. The authors recommended the use of HBOT in the management of intractable cyclophosphamide-induced HC as an effective and low-risk therapy.
Ravi and colleagues (2017) stated that radiation therapy for the treatment of head and neck cancer can injure normal tissues and have devastating side effects. Hyperbaric oxygen therapy is known to reduce the severity of radiation-induced injury by promoting wound healing.
Moran (2014) noted that digital ulcers are difficult to heal, increasing the chance of infection, gangrene, amputation and limited functional use of hands. They are a complication in scleroderma or systematic sclerosis (SSc) and occur in approximately 50% of patients. This was a systematic review of the evidence supporting the use of non-pharmaceutical therapeutic modalities and their effectiveness to facilitate the healing of chronic digital ulcers in patients with scleroderma. The author carried out a comprehensive review of computerized databases from 2000 to 2013: PubMed/Medline, CINAHL, Pedro, OT Seeker, OT Search, OVID, and Proquest as well as manual review of other resources using the following search terms scleroderma or systemic sclerosis and/or digital ulcers, specific modalities (low level laser therapy, electrical stimulation, intermittent compression, ultrasound, vitamin E, myofascial release, wound dressings, iontophoresis, negative pressure therapy, and exercise), chronic wounds, and wound care. English language studies, from 2000 to January 2013, which used therapeutic modalities to facilitate healing of digital ulcers and use healing of the digital ulcer as an outcome measure were reviewed. Of the 403 identified articles, only 11 studies addressed non-pharmaceutical treatment modalities to facilitate healing for digital ulcers. Exercise had no direct effect on healing ulcers. The following studies were positive but have limitations in design and sample size: HBOT (n = 2), negative pressure therapy (n = 1), intermittent compression (n = 27) and acoustic pressure wound healing (n = 1). Vitamin E gel showed a significant difference compared to a control group (n = 27). Iontophoresis studies have shown that the modality increases blood flow but the results in 5 different studies are mixed and the application and intensity were inconsistent. The author concluded that no one modality was proven to be the most effective; larger efficacy studies on treating digital ulcers are needed in order to develop appropriate care guidelines to improve outcomes, promote function and lower health-care costs.
On behalf of the American Academy of Otolaryngology-Head and Neck Surgery, Stachler and colleagues (2012) stated that sudden hearing loss (SHL) is a frightening symptom that often prompts an urgent or emergent visit to a physician. These investigators provided an evidence-based guideline for the diagnosis, management, and follow-up of patients who present with SHL. The guideline primarily focused on sudden sensorineural hearing loss (SSNHL) in adult patients (aged 18 and older). Prompt recognition and management of SSNHL may improve hearing recovery and patient quality of life (QOL). Sudden sensorineural hearing loss affects 5 to 20 per 100,000 population, with about 4,000 new cases per year in the United States. This guideline was intended for all clinicians who diagnose or manage adult patients who present with SHL. The panel recognized that patients enter the health care system with SHL as a non-specific, primary complaint. Therefore, the initial recommendations of the guideline deal with efficiently distinguishing SSNHL from other causes of SHL at the time of presentation. By focusing on opportunities for quality improvement, the guideline should improve diagnostic accuracy, facilitate prompt intervention, decrease variations in management, reduce unnecessary tests and imaging procedures, and improve hearing and rehabilitative outcomes for affected patients.
The therapy works by supersaturating the blood tissues with oxygen via increased atmospheric pressure as well as increased oxygen concentrations. Studies have demonstrated that this therapy increases the available oxygen to the body by 10 to 20 times normal levels.
The premise of HBOT is that the increased pressure results in increased oxygen levels in systemic circulation and the body’s tissues with the goal of improving healing of wounds, injuries or to support oxygen transport in acutely anemic or hypoxic individuals. Clinical Indications.
This document addresses the use of hyperbaric oxygen therapy (HBOT), which can be applied systemically, topically, or to one or more limbs alone. HBOT involves the use of pressurized room air, 100% oxygen, or room air enriched with a specific concentration of oxygen. The premise of HBOT is that the increased pressure results in increased oxygen ...
Pyoderma gangrenosum: A condition of the skin leading to open ulcers. Systemic hyperbaric oxygen: A therapy that involves sealing an individual inside a room or container, then exposing the individual to pure oxygen at greater than one atmosphere of pressure. Thermal: Related to heat.