Why ICD-10 codes are important
MEDICARE LOCAL COVERAGE ARTICLE (LCA) - A57718 PROCEDURE CODES: 82306, 82652 Vitamin D Assay Testing For services performed on or after 2-3-2017 DLS TEST CODE AND NAME The following ICD-10-CM codes support the medical necessity of procedure code 82652 The following ICD-10-CM codes support the medical necessity of procedure code 82306
Sequelae of unspecified nutritional deficiency
2. For 82306: If more than one LCD-listed condition contributes to Vitamin D deficiency in a given patient and/or is improved by Vitamin D administration, coders should use: ICD-10 E55. 9 UNSPECIFIED VITAMIN D DEFICIENCY. This code should not be used for any other indication.
ICD-10 | Vitamin D deficiency, unspecified (E55. 9)
ICD-10 code: E55. 9 Vitamin D deficiency, unspecified.
Vitamin D deficiency (ICD-10 CM Diagnosis Code E55. 9) is an overlooked diagnosis for a number of alarming symptoms and afflictions. Depression, fatigue, anxiety, kidney disease, weakened immune system, and even psoriasis have been known to manifest as a result of vitamin D deficiency.
ICD-10 code E56. 9 for Vitamin deficiency, unspecified is a medical classification as listed by WHO under the range - Endocrine, nutritional and metabolic diseases .
Measurement of 1, 25-OH Vitamin D level is indicated for patients with: unexplained hypercalcemia (suspected granulomatous disease or lymphoma) unexplained hypercalciuria (suspected granulomatous disease or lymphoma) suspected genetic childhood rickets.
ICD-10 code D51. 9 for Vitamin B12 deficiency anemia, unspecified is a medical classification as listed by WHO under the range - Diseases of the blood and blood-forming organs and certain disorders involving the immune mechanism .
This test is appropriate for assessment of vitamin D deficiency. Vitamin D, 1,25-dihydroxy (CPT code 82652) is primarily indicated during patient evaluations for hypercalcemia and renal failure.
Vitamin D deficiency may lead to a variety of disorders, the most infamous of which is rickets. Evaluating patients’ vitamin D levels is accomplished by measuring the level of 25-hydroxyvitamin D. Measurement of other metabolites is generally not medically necessary.
Vitamin D deficiency results in abnormalities in calcium, phosphorus, and bone metabolism. Specifically, vitamin D deficiency causes a decrease in the efficiency of intestinal calcium and phosphorus absorption of dietary calcium and phosphorus, resulting in an increase in parathyroid hormone (PTH) levels.
Vitamin D is called a “vitamin” because of its exogenous source, predominately from oily fish in the form of vitamin D2 and vitamin D3.
Vitamin D has been described as an immunomodulator targeting various immune cells, including monocytes, macrophages, T-lymphocytes, and B-lymphocytes.5 Studies have suggested that vitamin D plays an important role in maintenance of the immune system.
Once in the blood, vitamin D2 and D3 from diet or skin bind with vitamin D binding protein and are carried to the liver where they are hydroxylated to yield calcidiol. Calcidiol then is converted in the kidney to calcitriol by the action of 1a-hydroxylase (CYP27B1).
A major source of vitamin D for most humans comes from exposure of the skin to sunlight typically between 1000 hours and 1500 hours in the spring, summer, and fall. Vitamin D produced in the skin may last at least twice as long in the blood compared with ingested vitamin D.