The 2022 edition of ICD-10-CM M43.0 became effective on October 1, 2021.
Spondylolysis. M43.0 should not be used for reimbursement purposes as there are multiple codes below it that contain a greater level of detail. The 2021 edition of ICD-10-CM M43.0 became effective on October 1, 2020.
What is spondylolysis? Spondylolysis is a spinal fracture of a bone structure called the pars interarticularis, which connects the facet joints of the spine. The spinal cord is protected by rings of bone that make up the middle and posterior (rear) portion of the spinal column. In each of these rings, the pars interarticularis (or simply "pars" ...
However, spondylolisthesis can also be caused by the deterioration (degeneration) of the facet joints and discs of the spine, independent of any fracture. This is known as degnerative spondylolisthesis. Spondylolisthesis animation.
There are two operations that may be performed: A laminectomy, a type of spinal decompression surgeryin which the surgeon removes that portion of the vertebra that includes the pars interarticularis. This is used if there are nerve roots being affected that require decompression. A posterior lumbar fusion.
Spondylolisthesis is a condition where spinal vertebrae slip forward over one another. This is often caused when the crack of a pars fracture (spondylolysis ) affects both sides of connection between the lamina and pedicles. In this way, spondylolisthesis can be a direct result of spondylolysis.
The lower portion of the spinal column is called the lumbar spine. Spondylolysis most commonly occurs in the lower back in the lowest lumbar vertebra (L5). In athletes, this type of injury can be seen when the back is bent backwards repeatedly, in activities such as gymnastics, karate and football.
Evaluation for this condition would include a review of the patient’s medical history and a physical exam, followed by X-rays, which can detect pars fractures.
pain in the back, neck, legs, thighs or shoulder. muscle spasms or weakness. headache. tingling, numbness and/or stiffness in the back.
Radiographically visualized spondylolysis has an association with spondylolisthesis approximately 25% of the time[13]
Two common clinical presentations of a pars defect include the imaging of an asymptomatic adolescent or adult in whom there is the incidental discovery of a pars defect. The second common presentation is an adolescent athlete involved in a sport requiring repetitive lumbar loading in extension and rotation, presenting with acute or insidious onset low back pain that is aggravated by continued lumbar loading. Although this history is typical, there is a broad differential diagnosis that might explain these symptoms. As such, the diagnosis of a pars interarticularis defect confirmation is only with radiographic support. Depending on the time of presentation and degree of injury, most cases of pars defects respond well to conservative treatment and relative rest from sport.
Plain radiographs can miss some lesions, especially acute injury. Other useful imaging modalities include CT, MRI, and bone scan. A bone scan is the most sensitive modality, and best detects early pars defects. This test should be utilized when there is high clinical suspicion for pars lesion, and initial imaging studies are equivocal. After plain radiographs, the imaging modality of choice is either MRI without contrast or CT scan without contrast. CT scan is the best imaging tool for determining fracture size and extent and is the most appropriate modality for follow-up assessment of healing. CT has the downside of additional radiation exposure, which is particularly concerning in the pediatric & adolescent population. Similar to a bone scan, MRI can be useful for early detection of acute lesions by the presence of bone marrow edema on T2 weighted sequences. Additionally, MRI has the benefit of no radiation exposure. MRI is somewhat limited, though, in its ability to adequately depict the cortical integrity of incomplete fractures. [19]
The exact cause is still unclear. Currently, the most accepted theory is repetitive mechanical stress, specifically lumbar extension and rotation, which results in overuse or stress fracture to the pars interarticularis.[2] This theory garners support from the fact that, as noted below in epidemiology, the research observed zero cases of pars defects in 500 newborns and zero cases of pars defects in 143 non-ambulatory patients, suggesting this pathology develops as a result of repetitive axial loading over time. [1][3]Additionally, this theory is supported by the progression of unilateral pars defects into bilateral pars defects with age, again suggesting repetitive axial loading over time, both leading to the initial injury as well as disease progression.[4] As discussed, although generally thought to be the result of chronic repetitive stress to the pars region, these injuries can also occur due to a single acute overload injury. [5]
Pars defects (spondylolysis) subdivide into five categories according to the Wiltse-Newman Classification[22]:
The pars interarticularis is most susceptible to chronic axial loading injury because it is a weak point in the vertebrae, and this region bears the highest stress load in extension/flexion.[6] The weakness of the pars region is multifactorial, with a hereditary and an acquired mechanical component. Mechanical factors include the physically narrow structure of the pars interarticularis as compared to other regions of the vertebrae. Furthermore, the pars in the lower lumbar vertebra characteristically have uneven trabeculation and cortication. The inherent mechanical flaws of the pars interarticularis in combination with the high-stress loads seen in the lower lumbar region render this region prone to stress fractures.
Sagi et al. demonstrated, via histomorphic analysis, the pars interarticularis begins to ossify at 12 to 13 weeks gestation by endochondral ossification. In the lower lumbar vertebra, the ossification center originates in the pars region resulting in uneven distribution of trabeculation and cortication in this region. As a result of this uneven distribution of isthmic ossification, this region is potentially more susceptible to fatigue fracture. In contrast, in the upper lumbar vertebra, the ossification center arises at the end of the pedicle, which results in more uniform trabeculation throughout the pars. [14]