icd 10 code for pelvic prolapse syndrome

What is the ICd 9 code for pelvic pain?

For Pelvic Pain in Pregnancy ICD 9 code 646.83/625.9 should be used

What is pelvic pain syndrome?

Pelvic Pain Syndrome ICD 9,10 Codes. Pelvic pain is any pain in the area of the pelvis, which is the lower part of the abdomen between the hip bones. There are so many other terminologies for Pelvic pain. It is also known as abnormal development of female secondary sexual characteristics. Pelvic pain is also termed as female acute pelvic pain, ...

How to treat apical prolapse?

These investigators reviewed the therapeutic options for apical prolapse, specifically. Both conservative and surgical management options are acceptable and should be based on patient preferences. Pessaries are the most commonly used conservative management options. Guided pelvic floor muscle training is more beneficial than self-taught Kegel exercises, though may not be effective for high stage or apical prolapse. Surgical options include abdominal and vaginal approaches, the latter of which can be performed open, laparoscopically, and robotically. A systematic review has demonstrated that sacrocolpopexy has better long-term success for treatment of apical prolapse than vaginal techniques, but vaginal surgery can be considered an acceptable alternative. Recent data has demonstrated equal effectiveness between uterosacral ligament suspension and sacrospinous ligament suspension at 1 year. To-date, 2 randomized controlled trials (RCTs) have demonstrated equal effectiveness between robotic and laparoscopic sacrocolpopexy. Though abdominal approaches may have increased long-term durability, when counseling their patients, surgeons should consider longer operating times and increased pain and cost with these procedures compared to vaginal surgery.

What is MRI in organ prolapse?

Dynamic magnetic resonance imaging (MRI) in persons with complex organ prolapse to supplement the physical examination

What causes a vaginal collapse?

It is usually caused by weakness of the pelvic and vaginal tissues and muscles and may occur alone or along with prolapse of other pelvic organs.

What is sacrocolpopexy for?

Sacrocolpopexy for the treatment of vaginal apical prolapse repair

Is pelvic organ prolapse genetic?

Ward et al (2014) stated that given current evidence supporting a genetic predisposition for pelvic organ prolapse, they conducted a systematic review of published literature on the genetic epidemiology of pelvic organ prolapse. Inclusion criteria were linkage studies, candidate gene association and genome-wide association studies in adult women published in English and indexed in PubMed through December 2012, with no limit on date of publication. Methodology adhered to the PRISMA guidelines. Data were systematically extracted by 2 reviewers and graded by the Venice criteria for studies of genetic associations. A meta-analysis was performed on all SNPs evaluated by 2 or more studies with similar methodology. The meta-analysis suggested that collagen type 3 alpha 1 (COL3A1) rs1800255 genotype AA is associated with pelvic organ prolapse (OR, 4.79; 95 % CI: 1.91 to 11.98; p = 0.001) compared with the reference genotype GG in populations of Asian and Dutch women. There was little evidence of heterogeneity for rs1800255 (p value for heterogeneity = 0.94; proportion of variance because of heterogeneity, I(2) = 0.00 %). There was insufficient evidence to determine whether other SNPs evaluated by 2 or more papers were associated with pelvic organ prolapse. An association with pelvic organ prolapse was seen in individual studies for estrogen receptor alpha (ER-α) rs2228480 GA, COL3A1 exon 31, chromosome 9q21 (heterogeneity logarithm of the odds score 3.41) as well as 6 SNPs identified by a genome-wide association study. The authors concluded that overall, individual studies were of small sample size and often of poor quality. They stated that future studies would benefit from more rigorous study design as outlined in the Venice recommendations.

Can a sacrospinous ligament be combined with a vaginal hysterectomy?

Uterosacral ligament suspension (USLS) and sacrospinous ligament suspension (SSLS) are considered equally effective procedures and can be combined with a vaginal hysterectomy

Is urinary tract prolapse heritable?

Cartwright et al (2015) noted that family studies and twin studies demonstrated that lower urinary tract symptoms (LUTS) and pelvic organ prolapse are heritable. In this review, these investigators aimed to identify genetic polymorphisms tested for an association with LUTS or prolapse, and to assess the strength, consistency, and risk of bias among reported associations. PubMed and HuGE Navigator were searched up to May 1, 2014, using a combination of genetic and phenotype key words, including "nocturia", "incontinence", "overactive bladder", "prolapse", and "enuresis". Major genetics, urology, and gynecology conference abstracts were searched from 2005 through 2013. These researchers screened 889 abstracts, and retrieved 78 full texts. In all, 27 published and 7 unpublished studies provided data on polymorphisms in or near 32 different genes. Fixed and random effects meta-analyses were conducted using co-dominant models of inheritance. They assessed the credibility of pooled associations using the interim Venice criteria. In pooled analysis, the rs4994 polymorphism of the ADRB3 gene was associated with overactive bladder (OR, 2.5; 95 % CI: 1.7 to 3.6; n = 419). The rs1800012 polymorphism of the COL1A1 gene was associated with prolapse (OR, 1.3; 95 % CI: 1.0 to 1.7; n = 838) and stress urinary incontinence (OR, 2.1; 95 % CI: 1.4 to 3.2; n = 190). Other meta-analyses, including those for polymorphisms of COL3A1, LAMC1, MMP1, MMP3, and MMP9 did not show significant effects. Many studies were at high-risk of bias from genotyping error or population stratification. The authors concluded that these meta-analyses provided moderate epidemiological credibility for associations of variation in ADRB3 with overactive bladder, and variation of COL1A1 with prolapse. Moreover, they stated that clinical testing for any of these polymorphisms cannot be recommended based on current evidence.

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