D59.11Warm autoimmune hemolytic anemia D59. 11 is a billable/specific ICD-10-CM code that can be used to indicate a diagnosis for reimbursement purposes.
Warm autoimmune hemolytic anemia (WAHA) is an autoimmune disorder characterized by the premature destruction of healthy red blood cells (hemolysis). Autoimmune diseases occur when one's own immune system attacks healthy tissue.
AIHA caused by warm autoantibodies (w-AIHA), ie, antibodies that react with their antigens on the red blood cell optimally at 37°C, is the most common type, comprising ∼70% to 80% of all adult cases and ∼50% of pediatric cases.
A: ICD-10-CM code category D59. - (acquired hemolytic anemia) includes codes for hemolytic anemia from acquired conditions that result in the premature destruction of red blood cells, as opposed to hereditary disorders causing anemia.
Warm reactive autoantibodies are red blood cell (RBC) directed immune responses that are maximally reactive at 37°C. These are often associated with the destruction of red blood cells in vivo, causing hemolytic anemia in patients.
Warm antibody hemolytic anemia can often be differentiated from cold agglutinin disease by the temperature at which the direct antiglobulin test is positive; a test that is positive at temperatures ≥ 37° C indicates warm antibody hemolytic anemia, whereas a test that is positive at lower temperatures indicates cold ...
Warm autoantibodies are antibodies that react with intrinsic antigens on autologous RBCs at body temperature. Therefore, they are considered clinically significant, but the relationship between detection and pathologic effect is not well defined.
Any red cell antibody that binds its target antigen best at levels below body temperature (37 C) is commonly referred to as a “cold antibody” (this, of course, contrasts to “warm” antibodies that react best at or near body temperature).
Warm autoimmune hemolytic anemia (wAIHA) is caused by increased erythrocyte destruction by immunoglobulin G (IgG) autoantibodies, with or without complement activation. Antibody-dependent cell-mediated cytotoxicity by macrophages/activated lymphocytes occurs in the lymphoid organs and spleen (extravascular hemolysis).
Acquired autoimmune hemolytic anemia is a disorder that occurs in individuals who previously had a normal red blood cell system. The disorder may occur as the result of, or in conjunction with, some other medical condition, in which case it is “secondary” to another disorder.
Code D64. 9 is the diagnosis code used for Anemia, Unspecified, it falls under the category of diseases of the blood and blood-forming organs and certain disorders involving the immune mechanism. Anemia specifically, is a condition in which the number of red blood cells is below normal.
ICD-10 Code for Atherosclerotic heart disease of native coronary artery without angina pectoris- I25. 10- Codify by AAPC.
Warm autoimmune hemolytic anemia (wAIHA) is caused by increased erythrocyte destruction by immunoglobulin G (IgG) autoantibodies, with or without complement activation. Antibody-dependent cell-mediated cytotoxicity by macrophages/activated lymphocytes occurs in the lymphoid organs and spleen (extravascular hemolysis).
Two common causes of this type of anemia are sickle cell anemia and thalassemia. These conditions produce red blood cells that don't live as long as normal red blood cells.
What causes hemolytic anemia? Hemolytic anemia may be caused by inherited conditions that affect the red blood cells. It's also caused by certain infections or if someone receives a blood transfusion from a donor whose blood type didn't match.
Types of acquired hemolytic anemia include:immune hemolytic anemia.autoimmunehemolytic anemia (AIHA)alloimmune hemolytic anemia.drug-induced hemolytic anemia.mechanical hemolytic anemias.paroxysmal nocturnal hemoglobinuria (PNH)malaria, babesiosis and other infectious anemias.