icd 9 code for dysdiadochokinesia

Full Answer

What is another name for dysdiadochokinesis?

Other names. Dysdiadochokinesis, dysdiadokokinesia, dysdiadokokinesis. Specialty. Neurology. Dysdiadochokinesia ( DDK) is the medical term for an impaired ability to perform rapid, alternating movements (i.e., diadochokinesia ). Complete inability is called adiadochokinesia.

How is dysdiadochokinesia measured?

It is measurable by using the alternate motion rate (AMR). For example, speech AMR is measurable through verbal commands such as counting the number of syllable repetitions within a specific time (less than 1 minute).[1] Dysdiadochokinesia (diadochokinesia) is the inability to perform rapid alternating muscle movements.

Is dysdiadochokinesia a symptom of cerebellar dysfunction?

Dysdiadochokinesia is one of the features of cerebellar dysfunction, but other clinical findings associated with cerebellar dysfunction can help you localize and narrow the differential diagnosis of the patient. Clinical Symptoms

How is dysdiadochokinesia diagnosed in dysarthria?

Dysdiadochokinesia often presents in motor speech disorders (dysarthria), therefore testing for dysdiadochokinesia can be used for a differential diagnosis. Dysdiadochokinesia has been linked to a mutation in SLC18A2, which encodes vesicular monoamine transporter 2 (VMAT2).

How to exercise if you have DDK?

Also, make sure to exercise in a safe environment. Avoid exercising on hard surfaces that could lead to injury if you fall. Warm up your muscles before beginning these exercises.

What is DDK in physiotherapy?

Treatment often depends on finding the underlying cause. An important part of treatment is working with a physiotherapist, occupational therapist, or a speech pathologist.

How does DDK affect your body?

DDK may affect three major body areas: That means you may have symptoms in one or more of those areas. You may experience some or all of these symptoms if you have DDK: a change in balance and walking, including slowness, or awkward or rigid movements. A person with DDK may have difficulty rapidly turning their hand over several times ...

What is a DDK?

Dysdiadochokinesia (DDK) is the medical term used to describe difficulty performing quick and alternating movements, usually by opposing muscle groups. It’s pronounced “dis-di-ad-o-ko-ki-nee-ze-a.”. DDK is not a disease itself, but rather a symptom of an underlying health problem.

Where does DDK come from?

DDK most often comes from a disturbance in the cerebellum. The cerebellum is a large part of the brain that controls voluntary muscle movements, posture, and balance. It’s thought that people with DDK are unable to switch opposing muscle groups on and off in a coordinated manner.

Can you walk with DDK?

You’ll be asked to walk normally, and then walk heel to toe. A person with DDK will be unable to perform these tests in a correct or coordinated way. Your movements may be clumsy, unusual, or slowed. If symptoms are thought to be from a cerebral lesion, your doctor will order an MRI to detect and describe the lesion.

How is dysdiadochokinesia treated?

Treatment for DDK largely focuses on treating the underlying cause. Once the underlying condition is managed, providers may work on resolving the associated symptoms that one may experience with DDK. Oftentimes providers will encourage physical therapy, such as balance exercises, strength training, and other exercises to improve motor function. In some cases, speech and occupational therapy may also be beneficial.

What are the most important facts to know about dysdiadochokinesia?

Dysdiadochokinesia (DDK) refers to the inability to perform coordinated, rapid muscle movements. DDK is often a symptom related to an underlying problem impacting the cerebellum. The cerebellum is the portion of the brain that controls balance, coordination, and complex tasks. When this area of the brain is damaged, the individual may have difficulty with fast, alternating motions. Doctors can diagnose DDK using a variety of tests to assess motor function. They may also need to perform additional imaging and blood work to understand the underlying cause. Treatment will largely depend on resolving the underlying cause but may also include the management of symptoms through physical, occupational, or speech therapy.

What is DDK in a patient?

DDK is a feature of cerebellar ataxia, or impaired coordination, and can be the result of a variety of conditions affecting the cerebellum. Cerebellar dysfunction can either be acquired or genetic. Some acquired conditions that impact the cerebellum include metabolic dysfunction conditions (e.g.

How to diagnose DDK?

Diagnosis of DDK typically requires a physical examination to assess motor control and cerebellar function . Various tests that may be performed include rapid movement testing, the Romberg test, and the Stewart Holmes rebound test. Rapid movement tests can be performed in a variety of ways but most commonly, the individual will be asked to start with the palm of one hand on a flat surface. They will then be asked to flip their hand repeatedly, so the palm side of their hand moves from a downward-facing to an upward-facing position. Separately, the Romberg test requires the individual to stand with their heels together and eyes closed. If the individual has trouble balancing, it may indicate cerebellar dysfunction. Finally, the Stewart Holmes rebound test requires the individual to push against resistance, which is then suddenly removed. The examiner will assess how well the individual can control their movement after the resistance is taken away.

What is a DDK?

Dysdiadochokinesia ( DDK) refers to the inability to perform rapid, alternating movements, often due to an underlying cause. Ordinarily, individuals can rapidly switch between opposing movements in a coordinated fashion, like when unscrewing a light bulb. However, someone with DDK will find that their muscle response is slow or awkward, ...

What does it mean when you have DDK?

However, someone with DDK will find that their muscle response is slow or awkward, especially in the upper and lower limbs. This can include poor coordination while rapidly tapping one’s finger or foot, opening and closing one’s fist, and switching muscular positions from flexion (bent) to extension (straight).

Can a sedative cause cerebellar dysfunction?

Finally, deficiencies in vitamins B, E, and thiamine can also increase the risk of developing cerebellar dys function.

What is dysdiadochokinesia?

Dysdiadochokinesia (diadochokinesia) is the inability to perform rapid alternating muscle movements. These can be quick, synchronous, and can include pronation/supination, fast finger tapping, opening and closing of the fists, and foot tapping. It is an essential component to evaluate in patients suspected of having a cerebellar disease.  These activities correlate well with numerous lifelong measures of disability. It is a form of ataxia that leads to the loss of coordination of speech and limbs.  It is measurable by using the alternate motion rate (AMR). For example, speech AMR is measurable through verbal commands such as counting the number of syllable repetitions within a specific time (less than 1 minute).[1]

What is the inability to perform rapid alternating muscle movements?

Dysdiadochokinesia (diadochokinesia) or diadochokinesis is the inability to defined as the inability to perform rapid alternating muscle movements. These can be quick, synchronous, and can include pronation/supination, fast finger tapping, opening and closing of the fists, and foot tapping. It is an essential component to evaluate in patients suspected to have a cerebellar disease. It affects speech, upper and lower extremities. This activity reviews the definition, evaluation, and treatment of disorders that manifest with dysdiadochokinesia and highlights the role of the interprofessional team in evaluating and treating patients with these symptoms and how it can provide clues to a narrowed diagnosis.

What is psychogenic evaluation?

Psychogenic: Psychiatric evaluation to uncover conversion disorders or psychogenic dysdiadochokinesia

Can dysdiadochokinesia be present in children?

Dysdiadochokinesia can be present in small children without other focalizing neurological deficits or mental retardation. Children under the age of 13 years can present dysdiadochokinesia in 8 to 20% of the cases if tested.[25]  It is highest in the 7-8 year range. This presentation could be due to decreased myelination at younger ages with improved corticocerebellar connections later in life.

Is dysdiadochokinesia a clinical finding?

There are no known complications as dysdiadochokinesia is a clinical finding of cerebellar dysfunction.

Is dysdiadochokinesia evidence based?

No evidence-based study is specific for dysdiadochokinesia, however de pending on the etiology, an interdisciplinary team that consists of a neurologist, physical therapy, occupational therapy, speech therapy, social workers, nursing, mental health counselors, psychiatrist, and at times, geneticist should be consulted to provide the best quality of life for the patient. (Level V) To improve outcomes, prompt consultation with an interprofessional group of specialists is recommended.  Collaboration, shared decision-making, and communication are crucial elements for a good outcome.

Is ataxia a poor prognosis?

For example, acute traumatic, vascular, or fast progressing hereditary ataxias may have a poorer prognosis on elderly patients with multiple medical co-morbidities. Infectious, inflammatory, and metabolic etiologies can have a better prognosis if identified and diagnosed quickly and treated aggressively.

What are the signs of dysdiadochokinesia?

When testing for dysdiadochokinesia with speech the patient is asked to repeat syllables such as /pə/, /tə/, and /kə/; variation, excess loudness, and irregular articular breakdown are signs of dysdiadochokinesia.

Where can you find abnormalities in diadochokinesia?

Abnormalities in diadochokinesia can be seen in the upper extremity, lower extremity and in speech. The deficits become visible in the rate of alternation, the completeness of the sequence, and in the variation in amplitude involving both motor coordination and sequencing. Average rate can be used as a measure of performance when testing for dysdiadochokinesia.

Is dysdiadochokinesia a symptom of Friedreich's ataxia?

Dysdiadochokinesia is also seen in Friedreich's ataxia and multiple sclerosis, as a cerebellar symptom (including ataxia, intention tremor and dysarthria ). It is also a feature of ataxic dysarthria. Dysdiadochokinesia often presents in motor speech disorders ( dysarthria ), therefore testing for dysdiadochokinesia can be used for ...

Overview

Symptoms (780–789)

• 780 General symptoms
• 781 Symptoms involving nervous and musculoskeletal systems
• 782 Symptoms involving skin and other integumentary tissue
• 783 Symptoms concerning nutrition, metabolism and development

Nonspecific abnormal findings (790–796)

Ill-defined and unknown causes of morbidity and mortality (797–799)