The ICD-10-CM is a catalog of diagnosis codes used by medical professionals for medical coding and reporting in health care settings. The Centers for Medicare and Medicaid Services (CMS) maintain the catalog in the U.S. releasing yearly updates.
J81. 0, acute pulmonary edema.
Abstract. Flash pulmonary edema (FPE) is a general clinical term used to describe a particularly dramatic form of acute decompensated heart failure.
J81. 0 is a billable/specific ICD-10-CM code that can be used to indicate a diagnosis for reimbursement purposes.
Hypertensive flash pulmonary edema (FPE) results from rapid elevation of the left ventricular end-diastolic pressure. Absence of underlying valve disease or cardiomyopathy, it is usually caused by renal vascular disease.
Flash pulmonary edema (FPE), is rapid onset acute pulmonary edema. It is most often precipitated by acute myocardial infarction or mitral regurgitation, but can be also caused by aortic regurgitation, heart failure, or almost any cause leading to elevated left ventricular filling pressures.
Often, "flash" pulmonary edema is related to a sudden rise in left-sided intracardiac filling pressures in the setting of hypertensive emergency, acute ischemia, new onset tachyarrhythmia, or obstructive valvular disease.
ICD-10 code R60. 9 for Edema, unspecified is a medical classification as listed by WHO under the range - Symptoms, signs and abnormal clinical and laboratory findings, not elsewhere classified .
ICD-9 Code Transition: 786.5 Code R07. 9 is the diagnosis code used for Chest Pain, Unspecified. Chest pain may be a symptom of a number of serious disorders and is, in general, considered a medical emergency.
ICD-10 code E87. 70 for Fluid overload, unspecified is a medical classification as listed by WHO under the range - Endocrine, nutritional and metabolic diseases .
Pulmonary edema is often caused by congestive heart failure. When the heart is not able to pump efficiently, blood can back up into the veins that take blood through the lungs. As the pressure in these blood vessels increases, fluid is pushed into the air spaces (alveoli) in the lungs.
Many drugs — ranging from aspirin to illegal drugs such as heroin and cocaine — are known to cause pulmonary edema. Blood clot in the lungs (pulmonary embolism). A blood clot moving from the blood vessels in the legs to the lungs can cause pulmonary edema.
Noncardiogenic pulmonary edema is caused by changes in capillary permeability as a result of a direct or an indirect pathologic insult, while cardiogenic pulmonary edema occurs due to increased capillary hydrostatic pressure secondary to elevated pulmonary venous pressure.
Mechanisms for non-cardiogenic pulmonary edema include an increased capillary permeability and changes in pressure gradients within the pulmonary vasculature causing inflammation.
If the documentation is unclear, clarification would be needed. Although linking language is not required, it is best practice to link the etiology to acute pulmonary edema, leaving no question about its underlying cause and providers should be educated as such.
The onset of acute pulmonary edema often has a sudden onset, but it can be gradual as well. A patient with acute pulmonary edema typically demonstrates a variety of symptoms such as shortness of breath, especially while lying flat or with activity, wheezing, bilateral infiltrates on chest x-ray, a feeling of drowning, tachypnea, tachycardia, dizziness, restlessness, anxiety/agitation, frothy and/or pink tinged sputum, cyanosis and a variety of additional symptoms based on the underlying etiology.