If the AKI has progressed to ATN then the code N17.0 is reported and not the code default in the Alphabetic Index for AKI. Other terms that may be used to describe ATN could be renal tubular necrosis or tubular necrosis.
N17.1—Acute kidney failure with acute cortical necrosis. This isn’t as commonly documented as ATN but coders will see this. If the AKI has progressed to acute cortical necrosis then N17.1 is reported and not the code default in the Alphabetic Index for AKI.
Some of the most common specific guidelines that come to mind are sepsis, OB/delivery records and transplant complications. Let’s look at an example of each of these: Just in my experience, I think sepsis is one of the most common conditions that are associated with a diagnosis of AKI.
ICD-10 Code for Acute kidney failure with tubular necrosis- N17. 0- Codify by AAPC.
Acute tubular necrosis (ATN) is a kidney disorder involving damage to the tubule cells of the kidneys, which can lead to acute kidney failure. The tubules are tiny ducts in the kidneys that help filter the blood when it passes through the kidneys.
Acute tubular necrosis (ATN) is the most common cause of acute kidney injury (AKI) in the renal category (that is, AKI in which the pathology lies within the kidney itself). The term ATN is actually a misnomer, as there is minimal cell necrosis and the damage is not limited to tubules.
Etiologically, this common condition can be categorized as prerenal, intrinsic or postrenal. Most patients have pre-renal acute renal failure or acute tubular necrosis (a type of intrinsic acute renal failure that is usually caused by ischemia or toxins).
Acute tubular necrosis is usually diagnosed by a nephrologist (kidney specialist). The diagnosis is mainly clinical but can be guided by microscopic examination of your urine. A biopsy of the kidney tissue can be done in certain cases, especially when the diagnosis is uncertain.
ATN may be classified as either toxic or ischemic. Toxic ATN occurs when the tubular cells are exposed to a toxic substance (nephrotoxic ATN). Ischemic ATN occurs when the tubular cells do not get enough oxygen, a condition that they are highly sensitive and susceptible to, due to their very high metabolism.
The two major causes of AKI that occur in the hospital are prerenal disease and acute tubular necrosis (ATN). Together, they account for approximately 65 to 75 percent of cases of AKI. (See 'Frequency of prerenal disease and acute tubular necrosis as a cause of AKI' below.)
As a clinical condition characterized by an acute onset of kidney injury, the principal differential diagnosis of AIN is its differentiation from ATN.
The majority of patients recover from ATN with the renal failure phase typically lasting 7-21 days. However, depending on the severity of the initial insult, time to renal recovery can often be prolonged and patients may require dialysis for months.
Intravenous furosemide or bumetanide in a single high dose (ie, 100-200 mg of furosemide) is commonly used, although little evidence indicates that it changes the course of ATN. The drug should be infused slowly because high doses can lead to hearing loss. If no response occurs, the treatment should be discontinued.
Normally, your kidneys will be able to heal on their own. This means that your treatment will be focused on preventing the buildup of fluids and waste products in your body. Your treatment phase can last anywhere from a few days to over six weeks. It depends on how badly damaged your kidneys are.
The course of ATN can be divided into three phases:Onset or initiating phase. Lasting hours or days, this is the time from onset of the precipitating event (for example, toxin exposure) until tubular injury occurs.Maintenance phase. ... Recovery phase.
584.7 is a legacy non-billable code used to specify a medical diagnosis of acute kidney failure with lesion of renal medullary [papillary] necrosis. This code was replaced on September 30, 2015 by its ICD-10 equivalent.
The GEMs are the raw material from which providers, health information vendors and payers can derive specific applied mappings to meet their needs.
584.5 is a legacy non-billable code used to specify a medical diagnosis of acute kidney failure with lesion of tubular necrosis. This code was replaced on September 30, 2015 by its ICD-10 equivalent.
The GEMs are the raw material from which providers, health information vendors and payers can derive specific applied mappings to meet their needs.
584.7 is a legacy non-billable code used to specify a medical diagnosis of acute kidney failure with lesion of renal medullary [papillary] necrosis. This code was replaced on September 30, 2015 by its ICD-10 equivalent.
The GEMs are the raw material from which providers, health information vendors and payers can derive specific applied mappings to meet their needs.