Within 10 years of the diagnosis of cirrhosis, more than 50% of patients develop ascites[1]. The development of ascites is associated with a poor prognosis, with a mortality of 15% at one-year and 44% at five-year follow-up, respectively[2].
What can I do to help keep my cirrhosis from getting worse?
K74. 60 - Unspecified cirrhosis of liver. ICD-10-CM.
Hepatic failure, unspecified without coma K72. 90 is a billable/specific ICD-10-CM code that can be used to indicate a diagnosis for reimbursement purposes. The 2022 edition of ICD-10-CM K72. 90 became effective on October 1, 2021.
ICD-10 Code for Alcoholic cirrhosis of liver without ascites- K70. 30- Codify by AAPC.
Table 1CirrhosisPhysician Visit CodeOHIP: 571Decompensated CirrhosisHospital Diagnostic CodesICD-9: 456.0, 456.2, 572.2, 572.3, 572.4, 782.4, 789.5 ICD-10 : I85.0, I86.4, I98.20, I98.3, K721, K729, K76.6, K76.7, R17, R1813 more rows•Aug 22, 2018
821. Revised descriptor for ICD-10-CM diagnosis code Z77. 29.
A disorder characterized by replacement of the liver parenchyma with fibrous tissue and regenerative nodules. It is usually caused by alcoholisms, hepatitis b, and hepatitis c. Complications include the development of ascites, esophageal varices, bleeding, and hepatic encephalopathy.
Alcoholic liver disease, unspecified K70. 9 is a billable/specific ICD-10-CM code that can be used to indicate a diagnosis for reimbursement purposes. The 2022 edition of ICD-10-CM K70. 9 became effective on October 1, 2021.
ICD-10-CM Code for Unspecified cirrhosis of liver K74. 60.
K70. 31 - Alcoholic cirrhosis of liver with ascites | ICD-10-CM.
ICD-10-CM Code for Liver disease, unspecified K76. 9.
ICD-10-CM Code for Other cirrhosis of liver K74. 69.
Sensitivity analysis was conducted to assess the consistency of the above results. We compared the use of the random- and fixed-effects models for analysis of the studies and found that there were no obvious differences between them .3).
Certain metabolic disorders such as hereditary hemochromatosis, 1-antitrypsin deficiency, Wilsons disease, and hepatic porphyria are associated with high risk for the development of HCC. These hereditary diseases are known to promote hepatocarcinogenesis as a result of increased inflammation and hepatocellular damage .
There are three common presentations. Patients may present with malignant liver tumors found incidentally during investigation for unrelated symptoms or conditions. The most common focal mass in the liver is a hemangioma.
Portal hypertension results in an increase in hydrostatic pressure within the splanchnic bed. Decreased oncotic pressure caused by decreased protein synthesis may contribute to the condition.
The baselines of our included studies are summarized in Table . In general, this meta-analysis involved 5545 patients with a mean or median follow-up of 5 to 46 months. The PLT cut-off values ranged from 75 to 150 , which were near the cut-off point for thrombopenia . The qualities of the studies were moderate to high .
Two independent investigators performed a systematic search using the PubMed, EmBase, and ISI Web of Science databases with no language restrictions. Our core search consisted of the terms AND combined with the terms . In addition, we contacted authors if full text or crucial data were not available.
After calculating the total effect size using a random-effects model, we found that a low PLT level before treatment indicated a poor prognosis. The forest plot is shown in Figure , and the pooled estimator was stratified by survival .