Portal hypertension code for any associated complications, such as:; portal hypertensive gastropathy (K31.89) ICD-10-CM Diagnosis Code K74.69 [convert to ICD-9-CM] Other cirrhosis of liver
code for any associated complications, such as:; portal hypertensive gastropathy (K31.89) ICD-10-CM Diagnosis Code K74.69 [convert to ICD-9-CM] Other cirrhosis of liver
Since cirrhosis is the dominant cause of portal hypertension in the West, presentation will be that of cirrhosis with decompensation, or directly attributable to portal hypertension. This includes GI bleeding, ascites and edema, encephalopathy, or nonspecific constitutional symptoms (eg, fatigue, lethargy, anorexia).
Portal hypertension. Abnormal increase of resistance to blood flow within the hepatic portal system, frequently seen in liver cirrhosis and conditions with obstruction of the portal vein. Abnormally increased pressure in the portal venous system, frequently seen in cirrhosis of the liver and other conditions causing obstruction of the portal vein.
ICD-10 code K76. 6 for Portal hypertension is a medical classification as listed by WHO under the range - Diseases of the digestive system .
ICD-10-CM K76. 6 is grouped within Diagnostic Related Group(s) (MS-DRG v39.0): 441 Disorders of liver except malignancy, cirrhosis or alcoholic hepatitis with mcc. 442 Disorders of liver except malignancy, cirrhosis or alcoholic hepatitis with cc.
ICD-10-CM K74. 69 is grouped within Diagnostic Related Group(s) (MS-DRG v39.0): 432 Cirrhosis and alcoholic hepatitis with mcc. 433 Cirrhosis and alcoholic hepatitis with cc.
Your body carries blood to your liver through a large blood vessel called the portal vein. Cirrhosis slows your blood flow and puts stress on the portal vein. This causes high blood pressure known as portal hypertension.
Table 1ICD-10-AM coden with codeCirrhosisK70.3 Alcoholic cirrhosis of liver193K74.4 Secondary biliary cirrhosis*12K74.5 Biliary cirrhosis, unspecified617 more rows•Sep 17, 2020
Portal hypertension is a term used to describe elevated pressures in the portal venous system (a major vein that leads to the liver). Portal hypertension may be caused by intrinsic liver disease, obstruction, or structural changes that result in increased portal venous flow or increased hepatic resistance.
60 - Unspecified cirrhosis of liver.
Table 1CirrhosisPhysician Visit CodeOHIP: 571Decompensated CirrhosisHospital Diagnostic CodesICD-9: 456.0, 456.2, 572.2, 572.3, 572.4, 782.4, 789.5 ICD-10 : I85.0, I86.4, I98.20, I98.3, K721, K729, K76.6, K76.7, R17, R1813 more rows•Aug 22, 2018
K74.60 Unspecified cirrhosis of liver.K74.69 Other cirrhosis of liver.
The most common cause of portal hypertension is cirrhosis, or scarring of the liver. Cirrhosis results from the healing of a liver injury caused by hepatitis, alcohol abuse or other causes of liver damage. In cirrhosis, the scar tissue blocks the flow of blood through the liver and slows its processing functions.
With regard to the liver itself, causes of portal hypertension usually are classified as prehepatic, intrahepatic, and posthepatic.
Portal hypertension is elevated pressure in your portal venous system. The portal vein is a major vein that leads to the liver. The most common cause of portal hypertension is cirrhosis (scarring) of the liver.
Most clinical consequences of portal hypertension can be attributed to the development of portosystemic collateral vessels to return splanchnic blood to the heart. These vessels may form at several sites in the gut circulation. The most important are esophageal varices, formed by gross dilation of esophageal submucosal veins. These vessels carry blood from the coronary veins of the portal system into the azygos-hemiazygos veins. Other collateral sites include the umbilical vein into the omphalomesenteric vein. This occasionally results in striking dilation and prominence of the collateral vessels on the anterior abdominal wall with centrifugal radiation outward from the umbilicus, a pattern known as caput medusae. Rarely, a venous hum may be heard over such dilated veins (Cruveilhier’s sign). Other sites of collateral formation include the retroperitoneal cavity, splenorenal veins between the left kidney and the spleen, and vessels between the rectal and inferior mesenteric circulations. The latter results in large dilated, inferior rectal veins, often mistaken as gross hemorrhoids. Rarely, collaterals can develop in atypical sites (eg, duodenum, colon, or vagina). Portopulmonary collaterals have also been described.
In congestive gastropathy, mesenteric venous hypertension leads to congestion of the gastric mucosa, particularly the fundus, rendering it more susceptible to damage and bleeding even with minor insults (eg, modest alcohol or aspirin ingestion). Other causes of GI bleeding (eg, duodenal or gastric ulcer, Mallory-Weiss lacerations ) account for bleeding in the remaining patients.
Presinusoidal hypertension can be either intra- or prehepatic: Prehepatic (extrahepatic) causes include portal and splenic vein thromboses. Intrahepatic presinusoidal hypertension occurs in schistosomiasis, myelofibrosis and leukemic liver infiltration, idiopathic portal fibrosis, nodular regenerative hyperplasia, and granulomatous diseases (eg, sarcoidosis and early stages of primary biliary cirrhosis). In all cases of presinusoidal hypertension, the directly measured portal venous pressure will greatly exceed the hepatic venous pressure gradient, which should be normal or near normal. The presinusoidal block prevents transmission of the elevated portal pressure to the wedged hepatic vein. The overwhelming basis for sinusoidal and postsinusoidal portal hypertension is cirrhosis, particularly that due to alcohol. Postsinusoidal hypertension can again be divided into intrahepatic and posthepatic. Posthepatic causes include chronic heart failure, constrictive pericarditis, and obstruction of the hepatic venous outflow tract by membranous webs in the inferior vena cava. Intrahepatic postsinusoidal causes of portal hypertension include occlusive disease of the small veins and venules (veno-occlusive disease) and occlusions in large hepatic veins (Budd-Chiari syndrome). All cases of sinusoidal and postsinusoidal portal hypertension are associated with hepatic venous pressure gradients, which are about equal to the directly measured portal venous pressures. The resistance to flow extends from the hepatic venous system to the portal vein.
It is usually caused by a block in the blood flow through the liver due to cirrhosis (scarring) of the liver. Increased blood pressure in the portal venous system.
Other diseases of liver. Clinical Information. A disorder characterized by an increase in blood pressure in the portal venous system. Abnormal increase of resistance to blood flow within the hepatic portal system, frequently seen in liver cirrhosis and conditions with obstruction of the portal vein.
They must be used in conjunction with an underlying condition code and they must be listed following the underlying condition. alcoholic liver disease ( K70.-) amyloid degeneration of liver ( E85.-) toxic liver disease ( K71.-) A disorder characterized by an increase in blood pressure in the portal venous system.
In most cases the manifestation codes will have in the code title, "in diseases classified elsewhere.". Codes with this title are a component of the etiology/manifestation convention. The code title indicates that it is a manifestation code.